Na,K-ATPase Subunit Heterogeneity as a Mechanism for Tissue-Specific Ion Regulation
Article Properties
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LanguageEnglish
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DOI (url)
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Publication Date2005/09/01
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Journal
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Indian UGC (journal)
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Refrences116
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Citations203
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Gustavo Blanco
Cite
Blanco, Gustavo. “Na,K-ATPase Subunit Heterogeneity As a Mechanism for Tissue-Specific Ion Regulation”. Seminars in Nephrology, vol. 25, no. 5, 2005, pp. 292-03, https://doi.org/10.1016/j.semnephrol.2005.03.004.
Blanco, G. (2005). Na,K-ATPase Subunit Heterogeneity as a Mechanism for Tissue-Specific Ion Regulation. Seminars in Nephrology, 25(5), 292-303. https://doi.org/10.1016/j.semnephrol.2005.03.004
Blanco G. Na,K-ATPase Subunit Heterogeneity as a Mechanism for Tissue-Specific Ion Regulation. Seminars in Nephrology. 2005;25(5):292-303.
Journal Categories
Medicine
Internal medicine
Specialties of internal medicine
Diseases of the genitourinary system
Urology
Medicine
Medicine (General)
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Refrences
| Title | Journal | Journal Categories | Citations | Publication Date |
|---|---|---|---|---|
| Ensembl 2005 | Nucleic Acids Research |
| 224 | 2005 |
| Database resources of the National Center for Biotechnology Information | Nucleic Acids Research |
| 231 | 2005 |
Accumulation of βm, a structural member of X,K-ATPase β-subunit family, in nuclear envelopes of perinatal myocytes | American Journal of Physiology-Cell Physiology |
| 12 | 2004 |
Na+pump α2-isoform specifically couples to contractility in vascular smooth muscle: evidence from gene-targeted neonatal mice | American Journal of Physiology-Cell Physiology |
| 54 | 2004 |
Na,K‐ATPase α2 inhibition alters calcium responses in optic nerve astrocytes | Glia |
| 28 | 2004 |
Refrences Analysis
The category
Science: Biology (General) 81
is the most frequently represented among the references in this article. It primarily includes studies from Journal of Biological Chemistry The chart below illustrates the number of referenced publications per year.
Refrences used by this article by year
Citations
| Title | Journal | Journal Categories | Citations | Publication Date |
|---|---|---|---|---|
| Channelopathies in epilepsy: an overview of clinical presentations, pathogenic mechanisms, and therapeutic insights | Journal of Neurology |
| 2024 | |
| The effect of cytochrome c on Na,K-ATPase | Journal of Bioenergetics and Biomembranes |
| 2024 | |
| ATP1A1-linked diseases require a malfunctioning protein product from one allele | Biochimica et Biophysica Acta (BBA) - Molecular Cell Research |
| 2024 | |
The Healthy and Diseased Retina Seen through Neuron–Glia Interactions | International Journal of Molecular Sciences |
| 2024 | |
Novel compound heterozygous ATP1A2 variants in a patient with fetal akinesia/hypokinesia sequence | American Journal of Medical Genetics Part A |
| 1 | 2023 |
Citations Analysis
The category
Science: Biology (General) 78
is the most commonly referenced area in studies that cite this article. The first research to cite this article was titled The transcription factor CREMτ and cAMP regulate promoter activity of the Na,K‐ATPase α4 isoform and was published in 2006. The most recent citation comes from a 2024 study titled Channelopathies in epilepsy: an overview of clinical presentations, pathogenic mechanisms, and therapeutic insights. This article reached its peak citation in 2017, with 19 citations. It has been cited in 144 different journals, 21% of which are open access. Among related journals, the International Journal of Molecular Sciences cited this research the most, with 6 citations. The chart below illustrates the annual citation trends for this article.