Signaling mechanisms, interaction partners, and target genes of STAT6

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HEBENSTREIT, D, et al. “Signaling Mechanisms, Interaction Partners, and Target Genes of STAT6”. Cytokine &Amp; Growth Factor Reviews, vol. 17, no. 3, 2006, pp. 173-88, https://doi.org/10.1016/j.cytogfr.2006.01.004.
HEBENSTREIT, D., WIRNSBERGER, G., HOREJSHOECK, J., & DUSCHL, A. (2006). Signaling mechanisms, interaction partners, and target genes of STAT6. Cytokine &Amp; Growth Factor Reviews, 17(3), 173-188. https://doi.org/10.1016/j.cytogfr.2006.01.004
HEBENSTREIT D, WIRNSBERGER G, HOREJSHOECK J, DUSCHL A. Signaling mechanisms, interaction partners, and target genes of STAT6. Cytokine & Growth Factor Reviews. 2006;17(3):173-88.
Citations
Title Journal Journal Categories Citations Publication Date
MaiJiTong granule attenuates atherosclerosis by reducing ferroptosis via activating STAT6-mediated inhibition of DMT1 and SOCS1/p53 pathways in LDLR−/− mice Phytomedicine
  • Science: Botany: Plant ecology
  • Medicine: Therapeutics. Pharmacology
  • Medicine: Other systems of medicine
  • Medicine: Therapeutics. Pharmacology
  • Medicine: Public aspects of medicine: Toxicology. Poisons
2024
Autosomal Dominant STAT6 Gain of Function Causes Severe Atopy Associated with Lymphoma

Journal of Clinical Immunology
  • Medicine: Internal medicine: Specialties of internal medicine: Immunologic diseases. Allergy
8 2023
Eblasakimab, a novel IL-13 receptor alpha 1 monoclonal antibody, blocks STAT6 phosphorylation with low dose in human volunteers Clinical Immunology
  • Medicine: Internal medicine: Infectious and parasitic diseases
  • Medicine: Internal medicine: Specialties of internal medicine: Immunologic diseases. Allergy
  • Medicine: Internal medicine: Specialties of internal medicine: Immunologic diseases. Allergy
2 2023
A germline STAT6 gain-of-function variant is associated with early-onset allergies Journal of Allergy and Clinical Immunology
  • Medicine: Internal medicine: Infectious and parasitic diseases
  • Medicine: Internal medicine: Specialties of internal medicine: Immunologic diseases. Allergy
  • Medicine: Internal medicine: Specialties of internal medicine: Immunologic diseases. Allergy
  • Medicine: Internal medicine: Specialties of internal medicine: Immunologic diseases. Allergy
20 2023
Dihydroartemisinin Promotes Neural Stem Cell Differentiation into Oligodendrocytes via STAT6/Chi3l3 Signaling Pathway in Microglia

Journal of Biomedical Nanotechnology
  • Technology: Chemical technology
  • Technology: Chemical technology: Biotechnology
  • Technology: Electrical engineering. Electronics. Nuclear engineering: Materials of engineering and construction. Mechanics of materials
2023
Citations Analysis
Category Category Repetition
Medicine: Internal medicine: Specialties of internal medicine: Immunologic diseases. Allergy62
Medicine: Medicine (General)56
Science: Biology (General)40
Science: Chemistry: Organic chemistry: Biochemistry30
Science: Biology (General): Genetics30
Science: Biology (General): Cytology27
Medicine: Therapeutics. Pharmacology25
Medicine: Public aspects of medicine: Toxicology. Poisons23
Medicine: Internal medicine: Neoplasms. Tumors. Oncology. Including cancer and carcinogens20
Medicine18
Medicine: Internal medicine: Infectious and parasitic diseases11
Science: Science (General)10
Medicine: Medicine (General): Medical technology10
Science: Microbiology9
Medicine: Pathology9
Science9
Medicine: Internal medicine: Specialties of internal medicine: Diseases of the circulatory (Cardiovascular) system7
Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry6
Technology: Chemical technology: Biotechnology6
Medicine: Internal medicine: Specialties of internal medicine: Diseases of the digestive system. Gastroenterology6
Medicine: Internal medicine: Specialties of internal medicine: Diseases of the endocrine glands. Clinical endocrinology6
Medicine: Internal medicine: Specialties of internal medicine: Diseases of the blood and blood-forming organs6
Science: Physiology5
Science: Physics5
Science: Chemistry: Analytical chemistry5
Science: Chemistry5
Technology: Electrical engineering. Electronics. Nuclear engineering: Materials of engineering and construction. Mechanics of materials4
Medicine: Dermatology4
Technology: Chemical technology4
Science: Chemistry: General. Including alchemy4
Medicine: Surgery3
Science: Botany: Plant ecology2
Medicine: Other systems of medicine2
Medicine: Internal medicine2
Science: Zoology2
Medicine: Internal medicine: Specialties of internal medicine: Nutritional diseases. Deficiency diseases2
Medicine: Internal medicine: Special situations and conditions1
Medicine: Internal medicine: Medical emergencies. Critical care. Intensive care. First aid1
Medicine: Surgery: Anesthesiology1
Science: Chemistry: Physical and theoretical chemistry1
Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry: Neurology. Diseases of the nervous system1
Agriculture: Aquaculture. Fisheries. Angling1
Agriculture: Animal culture: Veterinary medicine1
Science: Biology (General): Reproduction1
Science: Human anatomy1
Science: Physiology: Animal biochemistry1
Science: Botany1
Technology: Home economics: Nutrition. Foods and food supply1
Agriculture: Animal culture1
Agriculture: Plant culture1
Medicine: Internal medicine: Specialties of internal medicine: Diseases of the genitourinary system. Urology1
Geography. Anthropology. Recreation: Environmental sciences1
Technology: Environmental technology. Sanitary engineering1
Science: Biology (General): Ecology1
Medicine: Nursing1
Medicine: Internal medicine: Specialties of internal medicine: Diseases of the respiratory system1
The category Medicine: Internal medicine: Specialties of internal medicine: Immunologic diseases. Allergy 62 is the most commonly referenced area in studies that cite this article. The first research to cite this article was titled Respiratory Syncytial Virus Synergizes with Th2 Cytokines to Induce Optimal Levels of TARC/CCL17 and was published in 2007. The most recent citation comes from a 2024 study titled MaiJiTong granule attenuates atherosclerosis by reducing ferroptosis via activating STAT6-mediated inhibition of DMT1 and SOCS1/p53 pathways in LDLR−/− mice. This article reached its peak citation in 2010, with 28 citations. It has been cited in 155 different journals, 17% of which are open access. Among related journals, the The Journal of Immunology cited this research the most, with 14 citations. The chart below illustrates the annual citation trends for this article.
Citations used this article by year