Fanconi anemia complementation group A cells are hypersensitive to chromium(VI)-induced toxicity.
Article Properties
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LanguageEnglish
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DOI (url)
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Publication Date2002/10/01
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Indian UGC (journal)
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Citations13
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Susan K Vilcheck Department of Pharmacology, Program in Molecular and Cellular Oncology, The George Washington University Medical Center, 2300 Eye Street NW, Washington, DC 20037, USA.
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Travis J O'Brien Department of Pharmacology, Program in Molecular and Cellular Oncology, The George Washington University Medical Center, 2300 Eye Street NW, Washington, DC 20037, USA.
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Daryl E Pritchard Department of Pharmacology, Program in Molecular and Cellular Oncology, The George Washington University Medical Center, 2300 Eye Street NW, Washington, DC 20037, USA.
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Linan Ha Department of Pharmacology, Program in Molecular and Cellular Oncology, The George Washington University Medical Center, 2300 Eye Street NW, Washington, DC 20037, USA.
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Susan Ceryak Department of Pharmacology, Program in Molecular and Cellular Oncology, The George Washington University Medical Center, 2300 Eye Street NW, Washington, DC 20037, USA.
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Jamie L Fornsaglio Department of Pharmacology, Program in Molecular and Cellular Oncology, The George Washington University Medical Center, 2300 Eye Street NW, Washington, DC 20037, USA.
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Steven R Patierno Department of Pharmacology, Program in Molecular and Cellular Oncology, The George Washington University Medical Center, 2300 Eye Street NW, Washington, DC 20037, USA.
Cite
Vilcheck, Susan K, et al. “Fanconi Anemia Complementation Group A Cells Are Hypersensitive to chromium(VI)-Induced Toxicity”. Environmental Health Perspectives, vol. 110, no. suppl 5, 2002, pp. 773-7, https://doi.org/10.1289/ehp.02110s5773.
Vilcheck, S. K., O’Brien, T. J., Pritchard, D. E., Ha, L., Ceryak, S., Fornsaglio, J. L., & Patierno, S. R. (2002). Fanconi anemia complementation group A cells are hypersensitive to chromium(VI)-induced toxicity. Environmental Health Perspectives, 110(suppl 5), 773-777. https://doi.org/10.1289/ehp.02110s5773
Vilcheck, Susan K, Travis J O’Brien, Daryl E Pritchard, Linan Ha, Susan Ceryak, Jamie L Fornsaglio, and Steven R Patierno. “Fanconi Anemia Complementation Group A Cells Are Hypersensitive to chromium(VI)-Induced Toxicity”. Environmental Health Perspectives 110, no. suppl 5 (2002): 773-77. https://doi.org/10.1289/ehp.02110s5773.
Vilcheck SK, O’Brien TJ, Pritchard DE, Ha L, Ceryak S, Fornsaglio JL, et al. Fanconi anemia complementation group A cells are hypersensitive to chromium(VI)-induced toxicity. Environmental Health Perspectives. 2002;110(suppl 5):773-7.
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Citations
| Title | Journal | Journal Categories | Citations | Publication Date |
|---|---|---|---|---|
Interactions between chromium species and DNA in vitro and their potential role in the toxicity of hexavalent chromium | Metallomics |
| 2 | 2023 |
| Homologous Recombination and Translesion DNA Synthesis Play Critical Roles on Tolerating DNA Damage Caused by Trace Levels of Hexavalent Chromium | PLOS ONE |
| 7 | 2016 |
| Gene–environment interactions in heavy metal and pesticide carcinogenesis | Mutation Research - Genetic Toxicology and Environmental Mutagenesis |
| 46 | 2014 |
| Chromium(VI) Causes Interstrand DNA Cross-Linking in Vitro but Shows No Hypersensitivity in Cross-Link Repair-Deficient Human Cells | Chemical Research in Toxicology |
| 13 | 2013 |
| Chromium genotoxicity: A double-edged sword | Chemico-Biological Interactions |
| 245 | 2010 |
Citations Analysis
The category
Medicine: Public aspects of medicine: Toxicology. Poisons 6
is the most commonly referenced area in studies that cite this article. The first research to cite this article was titled Fanconi anaemia proteins: Major roles in cell protection against oxidative damage and was published in 2003. The most recent citation comes from a 2023 study titled Interactions between chromium species and DNA in vitro and their potential role in the toxicity of hexavalent chromium. This article reached its peak citation in 2009, with 3 citations. It has been cited in 10 different journals, 10% of which are open access. Among related journals, the Mutation Research - Genetic Toxicology and Environmental Mutagenesis cited this research the most, with 3 citations. The chart below illustrates the annual citation trends for this article.