Effects of carbon tetrachloride on calcium homeostasis
Article Properties
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LanguageEnglish
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DOI (url)
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Publication Date1989/08/01
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Journal
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Indian UGC (journal)
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Refrences33
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Citations22
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Emanuele Albano
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Rita Carini
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Maurizio Parola
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Giorgio Bellomo
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Lucia Goria-Gatti
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Giuseppe Poli
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Mario U. Dianzani
Cite
Albano, Emanuele, et al. “Effects of Carbon Tetrachloride on Calcium Homeostasis”. Biochemical Pharmacology, vol. 38, no. 16, 1989, pp. 2719-25, https://doi.org/10.1016/0006-2952(89)90559-5.
Albano, E., Carini, R., Parola, M., Bellomo, G., Goria-Gatti, L., Poli, G., & Dianzani, M. U. (1989). Effects of carbon tetrachloride on calcium homeostasis. Biochemical Pharmacology, 38(16), 2719-2725. https://doi.org/10.1016/0006-2952(89)90559-5
Albano, Emanuele, Rita Carini, Maurizio Parola, Giorgio Bellomo, Lucia Goria-Gatti, Giuseppe Poli, and Mario U. Dianzani. “Effects of Carbon Tetrachloride on Calcium Homeostasis”. Biochemical Pharmacology 38, no. 16 (1989): 2719-25. https://doi.org/10.1016/0006-2952(89)90559-5.
Albano E, Carini R, Parola M, Bellomo G, Goria-Gatti L, Poli G, et al. Effects of carbon tetrachloride on calcium homeostasis. Biochemical Pharmacology. 1989;38(16):2719-25.
Journal Categories
Medicine
Public aspects of medicine
Toxicology
Poisons
Medicine
Therapeutics
Pharmacology
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Refrences
| Title | Journal | Journal Categories | Citations | Publication Date |
|---|---|---|---|---|
| Glucocorticoids activate a suicide process in thymocytes through an elevation of cytosolic Ca2+ concentration | Archives of Biochemistry and Biophysics |
| 337 | 1989 |
| Ethanolinduced mobilization of calcium by activation of phosphoinositide-specific phospholipase C in intact hepatocytes | Journal of Biological Chemistry |
| 1987 | |
Free-radical metabolism of carbon tetrachloride in rat liver mitochondria. A study of the mechanism of activation | Biochemical Journal |
| 56 | 1987 |
| The role of lipid peroxidation in liver damage | Chemistry and Physics of Lipids |
| 215 | 1987 |
| Cytosolic calcium after carbon tetrachloride, 1,1-dichloroethylene, and phenylephrine exposure | Biochemical Pharmacology |
| 30 | 1987 |
Refrences Analysis
The category
Science: Biology (General) 28
is the most frequently represented among the references in this article. It primarily includes studies from Biochemical Pharmacology and Journal of Biological Chemistry. The chart below illustrates the number of referenced publications per year.
Refrences used by this article by year
Citations
| Title | Journal | Journal Categories | Citations | Publication Date |
|---|---|---|---|---|
Myrica rubraExtracts Protect the Liver from CCl4-Induced Damage | Evidence-Based Complementary and Alternative Medicine |
| 5 | 2011 |
| Role of IL-6 trans-signaling in CCl4 induced liver damage | Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease |
| 32 | 2010 |
| Effective protection of Terminalia catappa L. leaves from damage induced by carbon tetrachloride in liver mitochondria | The Journal of Nutritional Biochemistry |
| 42 | 2006 |
| Inhibition of ursolic acid on calcium-induced mitochondrial permeability transition and release of two proapoptotic proteins | Biochemical and Biophysical Research Communications |
| 18 | 2005 |
Hepatoprotection of Oleanolic Acid is Related to Its Inhibition on Mitochondrial Permeability Transition | The American Journal of Chinese Medicine |
| 2005 |
Citations Analysis
The category
Medicine: Public aspects of medicine: Toxicology. Poisons 9
is the most commonly referenced area in studies that cite this article. The first research to cite this article was titled Three models of free radical-induced cell injury and was published in 1989. The most recent citation comes from a 2011 study titled Myrica rubraExtracts Protect the Liver from CCl4-Induced Damage. This article reached its peak citation in 1994, with 3 citations. It has been cited in 17 different journals, 5% of which are open access. Among related journals, the Hepatology cited this research the most, with 2 citations. The chart below illustrates the annual citation trends for this article.