The Immunology of Mucosal Models of Inflammation

Article Properties
  • Language
    English
  • Publication Date
    2002/04/01
  • Indian UGC (Journal)
  • Refrences
    213
  • Citations
    970
  • Warren Strober Mucosal Immunity Section, Laboratory of Clinical Investigation, NIAID, NIH, Bethesda, Maryland 20892-1890;Division of Gastroenterology, Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts 02115
  • Ivan J. Fuss Mucosal Immunity Section, Laboratory of Clinical Investigation, NIAID, NIH, Bethesda, Maryland 20892-1890;Division of Gastroenterology, Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts 02115
  • Richard S. Blumberg Mucosal Immunity Section, Laboratory of Clinical Investigation, NIAID, NIH, Bethesda, Maryland 20892-1890;Division of Gastroenterology, Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts 02115
Abstract
Cite
Strober, Warren, et al. “The Immunology of Mucosal Models of Inflammation”. Annual Review of Immunology, vol. 20, no. 1, 2002, pp. 495-49, https://doi.org/10.1146/annurev.immunol.20.100301.064816.
Strober, W., Fuss, I. J., & Blumberg, R. S. (2002). The Immunology of Mucosal Models of Inflammation. Annual Review of Immunology, 20(1), 495-549. https://doi.org/10.1146/annurev.immunol.20.100301.064816
Strober W, Fuss IJ, Blumberg RS. The Immunology of Mucosal Models of Inflammation. Annual Review of Immunology. 2002;20(1):495-549.
Journal Categories
Medicine
Internal medicine
Specialties of internal medicine
Immunologic diseases
Allergy
Description

Can insights from animal models redefine our understanding of inflammatory bowel diseases (IBDs)? This review examines murine models of mucosal inflammation, revealing how immune imbalances can lead to loss of tolerance for mucosal antigens, thus driving IBD pathogenesis. These models offer a new paradigm for comprehending the complexities of IBDs. The authors analyze the immunology of IBDs through the lens of murine models, highlighting their common features and individual capacities to provide insight into IBD pathogenesis. The review emphasizes that these models demonstrate various types of immune imbalance that can lead to loss of tolerance for mucosal antigens and thus inflammation centered in the gastrointestinal tract. The findings suggest that murine models provide a roadmap for defining the immunology of IBDs and identifying unexpected ways to treat these diseases. This comprehensive analysis could pave the way for novel therapeutic interventions.

Published in the Annual Review of Immunology, this paper directly addresses the journal's focus on current advances in immunological research. By analyzing murine models of inflammatory bowel diseases (IBDs), the review aligns perfectly with the journal's scope. The authors' insights contribute significantly to understanding IBD pathogenesis and potential therapeutic strategies, enhancing the field of immunology.

Refrences
Citations
Citations Analysis
The first research to cite this article was titled Cutting Edge: Inflammatory Signals Drive Organ-Specific Autoimmunity to Normally Cross-Tolerizing Endogenous Antigen and was published in 2002. The most recent citation comes from a 2024 study titled Cutting Edge: Inflammatory Signals Drive Organ-Specific Autoimmunity to Normally Cross-Tolerizing Endogenous Antigen . This article reached its peak citation in 2005 , with 75 citations.It has been cited in 354 different journals, 22% of which are open access. Among related journals, the Inflammatory Bowel Diseases cited this research the most, with 76 citations. The chart below illustrates the annual citation trends for this article.
Citations used this article by year