Beyond X-Chromosome Inactivation: The Oncogenic Facet of XIST in Human Cancers

Article Properties
  • Language
    English
  • DOI (url)
    10.15616/bsl.2019.25.2.113
  • Publication Date
    2019/06/30
  • Journal
    Biomedical Science Letters
  • Indian UGC (journal)
  • Refrences
    50
  • Hamadi Madhi Department of Anatomy, Embryology Laboratory, and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul 03722, Korea
  • Myoung Hee Kim Department of Anatomy, Embryology Laboratory, and Brain Korea 21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul 03722, Korea
Cite
Madhi, Hamadi, and Myoung Hee Kim. “Beyond X-Chromosome Inactivation: The Oncogenic Facet of XIST in Human Cancers”. Biomedical Science Letters, vol. 25, no. 2, 2019, pp. 113-22, https://doi.org/10.15616/bsl.2019.25.2.113.
Madhi, H., & Kim, M. H. (2019). Beyond X-Chromosome Inactivation: The Oncogenic Facet of XIST in Human Cancers. Biomedical Science Letters, 25(2), 113-122. https://doi.org/10.15616/bsl.2019.25.2.113
Madhi H, Kim MH. Beyond X-Chromosome Inactivation: The Oncogenic Facet of XIST in Human Cancers. Biomedical Science Letters. 2019;25(2):113-22.
Refrences
Title Journal Journal Categories Citations Publication Date
10.12659/MSM.905370
Long noncoding RNA XIST acts as an oncogene in non-small cell lung cancer by epigenetically repressing KLF2 expression Biochemical and Biophysical Research Communications
  • Science: Biology (General)
  • Science: Biology (General)
  • Science: Physics
  • Science: Biology (General)
  • Science: Chemistry: Organic chemistry: Biochemistry
  • Science: Biology (General)
  • Science: Chemistry: Organic chemistry: Biochemistry
 157 2016
10.1042/BSR20170696
Long Non-coding RNA XIST Promotes Glioma Tumorigenicity and Angiogenesis by Acting as a Molecular Sponge of miR-429 Journal of Cancer
  • Medicine: Medicine (General)
  • Medicine: Internal medicine: Neoplasms. Tumors. Oncology. Including cancer and carcinogens
 95 2017
LncRNA XIST accelerates cervical cancer progression via upregulating Fus through competitively binding with miR-200a Biomedicine & Pharmacotherapy
  • Medicine: Medicine (General)
  • Medicine: Therapeutics. Pharmacology
  • Medicine: Public aspects of medicine: Toxicology. Poisons
 96 2018