Advances in molecular pathology, diagnosis, and treatment of amyotrophic lateral sclerosis

Article Properties
  • Language
    English
  • Publication Date
    2023/10/27
  • Journal
    BMJ
  • Indian UGC (journal)
  • Refrences
    203
  • Citations
    3
  • Hristelina Ilieva
  • Mithila Vullaganti
  • Justin Kwan
Abstract
Cite
Ilieva, Hristelina, et al. “Advances in Molecular Pathology, Diagnosis, and Treatment of Amyotrophic Lateral Sclerosis”. BMJ, 2023, p. e075037, https://doi.org/10.1136/bmj-2023-075037.
Ilieva, H., Vullaganti, M., & Kwan, J. (2023). Advances in molecular pathology, diagnosis, and treatment of amyotrophic lateral sclerosis. BMJ, e075037. https://doi.org/10.1136/bmj-2023-075037
Ilieva, Hristelina, Mithila Vullaganti, and Justin Kwan. “Advances in Molecular Pathology, Diagnosis, and Treatment of Amyotrophic Lateral Sclerosis”. BMJ, 2023, e075037. https://doi.org/10.1136/bmj-2023-075037.
Ilieva H, Vullaganti M, Kwan J. Advances in molecular pathology, diagnosis, and treatment of amyotrophic lateral sclerosis. BMJ. 2023;:e075037.
Refrences
Title Journal Journal Categories Citations Publication Date
Sensitivity of electrophysiological tests for upper and lower motor neuron dysfunction in ALS: A six‐month longitudinal study

Muscle & Nerve
  • Medicine
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry: Neurology. Diseases of the nervous system
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry
21 2010
MR imaging and localized proton spectroscopy of the precentral gyrus in amyotrophic lateral sclerosis 2000
Evidence for polygenic and oligogenic basis of Australian sporadic amyotrophic lateral sclerosis Journal of Medical Genetics
  • Medicine: Medicine (General): Medical technology
  • Science: Biology (General): Genetics
  • Science: Biology (General): Genetics
  • Medicine: Medicine (General)
2020
10.21203/rs.3.rs-386600/v1
White matter alterations differ in primary lateral sclerosis and amyotrophic lateral sclerosis Brain
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry: Neurology. Diseases of the nervous system
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry
91 2011
Citations
Title Journal Journal Categories Citations Publication Date
Fluid biomarkers for amyotrophic lateral sclerosis: a review

Molecular Neurodegeneration
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry: Neurology. Diseases of the nervous system
  • Medicine: Internal medicine: Special situations and conditions: Geriatrics
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry
1 2024
Stathmin 2 is a potential treatment target for TDP-43 proteinopathy in amyotrophic lateral sclerosis Translational Neurodegeneration
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry: Neurology. Diseases of the nervous system
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry
  • Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry
2024
Recent Research Trends in Neuroinflammatory and Neurodegenerative Disorders

Cells
  • Science: Biology (General): Cytology
  • Science: Biology (General)
  • Science: Chemistry: Organic chemistry: Biochemistry
  • Science: Biology (General): Cytology
2024
Citations Analysis
The category Medicine: Internal medicine: Neurosciences. Biological psychiatry. Neuropsychiatry: Neurology. Diseases of the nervous system 2 is the most commonly referenced area in studies that cite this article. The first research to cite this article was titled Stathmin 2 is a potential treatment target for TDP-43 proteinopathy in amyotrophic lateral sclerosis and was published in 2024. The most recent citation comes from a 2024 study titled Stathmin 2 is a potential treatment target for TDP-43 proteinopathy in amyotrophic lateral sclerosis. This article reached its peak citation in 2024, with 3 citations. It has been cited in 3 different journals, 100% of which are open access. Among related journals, the Translational Neurodegeneration cited this research the most, with 1 citations. The chart below illustrates the annual citation trends for this article.
Citations used this article by year