Deficiency of Pink1 promotes the differentiation of Th1 cells

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Mei, Xiaole, et al. “Deficiency of Pink1 Promotes the Differentiation of Th1 Cells”. Molecular Immunology, vol. 160, 2023, pp. 23-31, https://doi.org/10.1016/j.molimm.2023.06.005.
Mei, X., Lei, Y., Ouyang, L., Zhao, M., & Lu, Q. (2023). Deficiency of Pink1 promotes the differentiation of Th1 cells. Molecular Immunology, 160, 23-31. https://doi.org/10.1016/j.molimm.2023.06.005
Mei X, Lei Y, Ouyang L, Zhao M, Lu Q. Deficiency of Pink1 promotes the differentiation of Th1 cells. Molecular Immunology. 2023;160:23-31.
Refrences
Title Journal Journal Categories Citations Publication Date
OXPHOS promotes apoptotic resistance and cellular persistence in T H 17 cells in the periphery and tumor microenvironment

Science Immunology
  • Medicine: Internal medicine: Specialties of internal medicine: Immunologic diseases. Allergy
28 2022
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  • Science: Biology (General)
  • Science: Biology (General): Cytology
  • Science: Biology (General): Cytology
  • Science: Biology (General): Genetics
57 2021
Emerging views of mitophagy in immunity and autoimmune diseases Autophagy
  • Science: Biology (General): Genetics
  • Science: Biology (General): Cytology
247 2020
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  • Science: Science (General)
298 2019
Distinct modes of mitochondrial metabolism uncouple T cell differentiation and function Nature
  • Science: Science (General)
141 2019
Citations
Title Journal Journal Categories Citations Publication Date
The major histocompatibility complex participates in Parkinson’s disease Pharmacological Research
  • Medicine: Public aspects of medicine: Toxicology. Poisons
  • Medicine: Therapeutics. Pharmacology
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The category Medicine: Public aspects of medicine: Toxicology. Poisons 1 is the most commonly referenced area in studies that cite this article.